Axon degeneration from NMN accumulation can be prevented by bypassing the NAD+ salvage pathway (which is associated with the production of NMN) by providing nicotinic acid riboside (a precursor to nicotinic acid mononucleotide) as substrate for the body to make NAD+ via the primary Preiss Handler pathway instead.
Most "niacin" products are niacinamide. If you want the form discussed in this research, look for nicotinic acid specifically.
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