Studies tagged with: GPR109A

• Emerging roles of GPR109A in regulation of neuroinflammation in neurological diseases and pain
• Deficiency of metabolite sensing receptor HCA2 impairs the salutary effect of niacin in hemorrhagic shock
• Inflammation stimulates niacin receptor (GPR109A/HCA2) expression in adipose tissue and macrophages
• Activation of Gpr109a, receptor for niacin and the commensal metabolite butyrate, suppresses colonic inflammation and carcinogenesis
• Activation of Gpr109a, Receptor for Niacin and the Commensal Metabolite Butyrate, Suppresses Colonic Inflammation and Carcinogenesis
• Anti-inflammatory effects of nicotinic acid
• Decreased expression of G-protein-coupled receptors GPR43 and GPR109a in psoriatic skin can be restored by topical application of sodium butyrate
• Melatonin alleviates titanium nanoparticles induced osteolysis via activation of butyrate/GPR109A signaling pathway
• Activated niacin receptor HCA2 inhibits chemoattractant-mediated macrophage migration via Gβγ/PKC/ERK1/2 pathway and heterologous receptor desensitization
• A novel treatment target for Parkinson's disease
• Activation of the receptor (Gpr109a) for niacin and the commensal metabolite butyrate suppresses colonic inflammation and carcinogenesis
• Niacin fine-tunes energy homeostasis through canonical GPR109A signaling